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Inhibition of oxidases reduces superoxide formation and protects against alcohol-induced neuronal death [133]

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45 The increased G6PD reductive activity and its overall cytoplasmic level in Alzheimer's brain is a result of homeostasis towards counterbalancing the oxidative stress and neuro-degeneration induced by amyloid-β peptide.46, 47, 48 However, the amyloid-β peptide does not directly induce the activity of G6PD in AD