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Further, the over-expression of SOCS3 inhibits triple negative breast cancer (TNBC) growth and the formation of metastasis in mouse xenograft models, and its loss implies risk of relapses in TNBC patients [41]
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This multi-target mechanism explains why it has documented effects on skin, hair, wounds, lungs, and even cancer gene regulation — something that would be puzzling if it only had one mechanism
